MOTS-C
MS10 · 10mg · 2026-10-21 · N/A
COA PDF ↗
MS20Images are illustrative. Actual specifications are as selected and confirmed in writing.
Longevity & Mitochondrial Research
MOTS-C
Metabolism, insulin sensitivity and exercise capacity
MOTS-C specifications and reference prices. Select a specification to add it to your cart.
20mg/vial x 10vials
Reference priceUS$257.58
For laboratory research only. Not for human or veterinary use.
COA 检测报告 / Reports
MOTS-C
MS20 · 20mg · 2026-09-07 · N/A
COA PDF ↗Compare all specifications & reference prices
| SKU | Specification / pack | Reference price (USD) |
|---|---|---|
| MS10 | 10mg/vial x 10vials | US$154.55 |
| MS15 | 15mg/vial x 10vials | US$206.06 |
| MS20 | 20mg/vial x 10vials | US$257.58 |
| MS40 | 40mg/vial x 10vials | US$466.67 |
Temporarily sold-out specifications can still be added to an inquiry to ask about restocking. An inquiry is not an order or an agreed price.
DOCUMENTATION / MS20
MOTS-C — supplied COA and inquiry notes
The MOTS-C catalog group currently lists 4 specifications. Use the selector and specification table above to compare SKUs and reference prices. The linked report identifies sample MS20; its sample code is not a separate specification selection.
- Report sample code
- MS20
- Reported date
- 2026-09-07
- Sample purity (HPLC-UV)
- 99.61%
- Sample net content
- 21.53 mg
- Lot / batch field
- N/A
- Endotoxin result
- N/A
These values describe the supplied test sample only.
When inquiring about MOTS-C, include the exact SKU, pack quantity and destination. Ask for stock and batch-document confirmation. Temporarily sold-out specifications can still be queried; the cart is not a confirmed order.
For laboratory research only. Not for human or veterinary use. The supplied report footnotes also exclude diagnostic, therapeutic and clinical applications.
Explore Longevity & Mitochondrial Research ↗ · View all products
RESEARCH FOCUS
Main Uses & FAQs
This page organizes directly relevant information on MOTS-C regarding metabolism, insulin sensitivity, and exercise capacity. First, look at the research questions and findings, then verify the population or species, study preparations, and scope of interpretation.
Main TopicsMetabolism · Insulin Sensitivity · Exercise Capacity
References
Read the Full Research Review ↗Research SummaryKey Findings
What research findings exist for MOTS-C regarding metabolism, insulin sensitivity, and exercise performance?
MOTS-c enhances muscle mitochondrial bioenergetic performance and reduces reactive oxygen species-related damage, acting via PGC-1α and AMPK. Other studies report that MOTS-c increases exercise capacity in mice of various ages and regulates skeletal muscle metabolism.[1] ↗ [2] ↗
Before ReadingStudy populations, species, formulations and delivery conditions each have a defined scope. Research on an ingredient does not establish that this site's product batches have been validated for efficacy in humans or veterinary use.
Human observation + animal intervention2026REF. 01
What changes occur in skeletal muscle mitochondrial bioenergetics?
Research Findings
MOTS-c enhances muscle mitochondrial bioenergetic performance and reduces reactive oxygen species-related damage, acting via PGC-1α and AMPK.[1] ↗
Study Design
Transgenic mouse administration and skeletal muscle mitochondrial experiments; also observed in human exercise
Scope & Limitations
Mainly mechanism and animal indicators; does not prove weight loss or exercise efficacy in humans.
View the Article Title and Full Citation＋
MOTS-c improves intrinsic muscle mitochondrial bioenergetic health and efficiency in a PGC-1α/AMPK-dependent manner.
Free Radical Biology & Medicine · Volume and Issue Year 2026
First published 2026-01-09 · PMID 41520850
DOI 10.1016/j.freeradbiomed.2026.01.002
Animal administration + human exercise observation2021 · Historical basic researchREF. 02
How does the exercise ability of mice of different ages change?
Research Findings
MOTS-c increases exercise capacity in mice of various ages and regulates skeletal muscle metabolism.[2] ↗
Study Design
Administration to mice of different ages; others observed endogenous levels of exercise in humans.
Scope & Limitations
The human body is an endogenous expression induced by exercise and cannot be stated as having improved exercise performance through human injection.
View the Article Title and Full Citation＋
MOTS-c is an exercise-induced mitochondrial-encoded regulator of age-dependent physical decline and muscle homeostasis.
Nature communications · Volume/Issue Year 2021
First published 2021-01-20 · PMID 33473109
DOI 10.1038/s41467-020-20790-0
Animal / experimental studies2015 · Historical basic researchREF. 03
What changes occur in obesity and insulin resistance in the high-fat diet model?
Research Findings
MOTS-c prevents high-fat diet–related obesity and insulin resistance in mice, directly corresponding to metabolic research.[3] ↗
Study Design
Mouse administration and cellular mechanism; 2015 historical research
Scope & Limitations
No. Not clinical evidence for human weight loss; older studies, do not claim the latest progress.
View the Article Title and Full Citation＋
The mitochondrial-derived peptide MOTS-c promotes metabolic homeostasis and reduces obesity and insulin resistance.
Cell metabolism · Volume and issue year 2015
First published on 2015-03-03 · PMID 25738459
DOI 10.1016/j.cmet.2015.02.009
Currently, 3 original studies related to the theme of this page have been selected. The number of studies includes different designs, populations, or preparations, not commercial clinical efficacy ratings. This page is a usage-oriented positive data selection, not a systematic review or dosing recommendation. Historical studies, combination preparations, and special models are all separately indicated.
GO DEEPER
↗What Questions Do These Studies Address?
Compare study populations, findings and scope.
MOTS-C · Frequently Asked Questions.
What research findings exist for MOTS-C regarding metabolism, insulin sensitivity, and exercise performance?＋
MOTS-c enhances muscle mitochondrial bioenergetic performance and reduces reactive oxygen species-related damage, acting via PGC-1α and AMPK. Other studies report that MOTS-c increases exercise capacity in mice of various ages and regulates skeletal muscle metabolism.[1] ↗ [2] ↗
Does human exercise observation with MOTS-C equal effective human administration?＋
Not equal. In relevant studies, part of the human observation involved exercise-induced endogenous MOTS-c; evidence for improving exercise capacity comes from mice, and the two cannot be combined to claim ‘effective injection in humans’.[2] ↗
MOTS-C—these conclusions apply to which study populations?＋
Transgenic mouse administration and skeletal muscle mitochondrial experiments; also observed in human exercise; administration in mice of different ages; additional human exercise endogenous level observation; mouse administration and cellular mechanisms; 2015 historical study. Mainly mechanism and animal indicators; does not prove human weight loss or exercise efficacy.[1] ↗ [2] ↗ [3] ↗
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